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COVID-19 presents a wide range of symptoms, primarily affecting the respiratory system but also impacting other systems such as the cardiovascular and nervous systems.
Over time, extensive research has been conducted on the disease. While our understanding is still evolving, COVID-19 has been strongly linked to blood clotting disorders, leading primarily to venous but also arterial thrombosis. These complications contribute to worse outcomes and increased mortality. The underlying mechanism involves significantly elevated levels of inflammation and coagulation activity, referred to as thrombo-inflammation. This process is believed to cause small blood clots, which can lead to respiratory failure and the need for mechanical ventilation.
Endothelial cells appear to be a primary target of COVID-19. Endothelial damage and its associated structural and functional changes play a crucial role in the hyperinflammatory response triggered by the virus, which can result in severe disease progression. On one hand, the virus itself induces blood clots, increasing disease severity. On the other hand, prolonged hospitalization, immobility, ICU admission, underlying serious health conditions, and advanced age further elevate the risk of thrombosis.
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